湯偉晉 管理的部落格

2024年5月30日 星期四

Mitochondrial Glutathione in Cellular Redox Homeostasis and Disease Manifestation

Mitochondrial Glutathione in Cellular Redox Homeostasis and Disease Manifestation

Mitochondrial Glutathione in Cellular Redox Homeostasis and Disease Manifestation PY2024 IR95 IR96 Taiwan



Mitochondrial Glutathione in Cellular Redox Homeostasis and Disease Manifestation PY2024 IR95 IR96 Taiwan

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2024-05-31
Mitochondrial Glutathione in Cellular Redox Homeostasis and Disease Manifestation

Source or References (
資訊來源或是參考的資訊):
https://www.mdpi.com/1422-0067/25/2/1314
Info cited on 2024-05-31-WD5 (
資訊引用於 中華民國113年西元2024531) by 湯偉晉 (WeiJin Tang)
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Mitochondrial Glutathione in Cellular Redox Homeostasis and Disease Manifestation PY2024 IR95 IR96 Taiwan

Figure 5. The synthesis of glutathione requires the expression of three genes, namely, GCLC, GCLM, and GSS, in different tissues. These genes (GCLC, GCLM, and GSS) encode the GCL holoenzyme and glutathione synthetase. An overview of the RNA expression reveals that the RNA sequencing (RNA-SEQ) data are a combination of information from the Human Protein Atlas RNA-SEQ data and an internally generated consensus data combination. These datasets were obtained from the Human Protein Atlas Project (https://www.proteinatlas.org (accessed on 6 January 2024). GCLM, glutamate–cysteine ligase regulatory subunit; GCLC, glutamate–cysteine ligase catalytic subunit; GSS, glutathione synthetase; nTPM, transcripts per million.

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